THE ROLE OF INFLAMMATION FACTORS IN THE MECHANISMS OF PROTECTION AND DESTRUCTION OF PERIODONTAL TISSUES
Keywords:
part, body, reactions, acquiredAbstract
The occurrence and course of any disease of an infectious nature is determined by the strength of the body's response [18, 16]. The nature of this response depends on the antigenic specificity of previously acquired immunity and is largely determined by the human genotype [10, 16]. At the same time, only a part of the observed reactions of the body to infection can be considered as protective
References
Aimadinova, N.K. Early diagnosis of the development of periodontitis by determining prognostically significant RNA markers. / N.K. Aimadinova // Dentistry. - 2015. - No. 6. - T.94. - S. 54 - 55.
Aimadinova, N.K. The role of Prevotella intermedia in the course of chronic periodontitis, studied using real-time PCR / N.K. Aimadinova // VII Scientific-practical conference of young scientists "Fundamental and applied problems of dentistry and maxillofacial surgery". - Moscow, 2016.
Atrushkevich, V.G. Diagnosis and treatment of periodontal diseases in violation of mineral metabolism: Ph.D. dis. ... Dr. med. Sciences: 14.00.14. / Atrushkevich Victoria Gennadievna. - M., 2010. - 50 p.
Barer, G. The role of interferon and other cytokines in the occurrence and development of periodontal disease. / G. Barer, S. Grigoryan, N. Postnova // Cathedra. - 2006. - V. 5, No. 3. - S. 54-60.
Boriskina, O.A. Detection of prognostically significant molecular genetic markers for early high-precision diagnosis of the development of aggressive periodontitis: dis. ... cand. honey. Sciences: 14.00.14. / Boriskina Olga Andreevna. - M., 2014. - 146 p.
Ivanyushko, T.P. The study of opportunistic microorganisms by real-time PCR in patients with periodontitis. / T.P. Ivanyushko, L.V. Tumbinskaya, A.E. Donnikov // Dentistry. - 2011. - No. 5. - P.22-26.
Kozlov, S.A. Correction of the balance of the oral cavity as a condition for achieving an antiseptic effect in patients with inflammatory periodontal diseases. / S.A. Kozlov, A.N. Seleznev, L.N. Kolobkova and others // Bulletin of the Peoples' Friendship University of Russia. - 2007. - No. 2. - P.77-80.
Kofiadi, I.A. Methods detection oligonucleotide
polymorphisms: allele-specific PCR and hybridization with an oligonucleotide probe. / I.A. Kofiadi, D.V. Rebrikov // Genetics. - 2006. - V.42, No. 1. - P.22-32.
Tsepov, L.M. Factors of local resistance and immunological
oral reactivity. Methods for their clinical and laboratory assessment (literature review). 4.II. / L.M. Tsepov, L.Yu. Orekhova, A.I. Nikolaev and others //
Periodontology. - 2005. - No. 3 (36). - P.35-39.
Tsimbalistov, A.V. Features of the microflora of periodontal pockets in aggressive forms of periodontitis. / A.V. Tsimbalistov, T.T. Natsvlishvili, T.I. Kadurina and others // Institute of Dentistry. - 2010. - No. 4. - P.73-74.
Chepurkova, O.A. Features of microbiocenosis of the periodontal pocket in generalized periodontitis of moderate severity. / O.A. Chepurkova, M.G. Chesnokova, V.B. Nedoseko // Institute of Dentistry. - 2007. - No. 3. - P.86-88.
Amano, A. Variations of Porphyromonas gingivalis fimbriae in relation to microbial pathogenesis. / A. Amano, L. Nakagawa, N. Okahashi et al. // J. Periodont. Res. - 2004. - Vol.39. - P.136-142.
Anovazzi, G. Interleukin 4 haplotypes of susceptibility to chronic periodontitis are associated with IL-4 protein levels but not with clinical outcomes of periodontal therapy. / G. Anovazzi, LS Finoti, SC Corbi et al. // Hum. Immunol. - 2013. - Vol.74(12). - P.1688-95.
Bourmenskaya, O. Host gene expression profiling of cervical smear is eligible for cancer risk evaluation. / O. Bourmenskaya, E. Shubina, D. Trofimov et al. // J. Clin. Pathol. - 2013. - Vol.66 (4). - P.282-285.
Braga, RR Quantification of five putative periodontal pathogens in female patients with and without chronic periodontitis by real-time polymerase chain reaction. / RR Braga, MA Carvalho, O. Bruna-Romero et al. // Anaerobe. - 2010. - Vol.16(3). - P.234-239.
Burke, W. Genomics as a probe for disease biology. / W. Burke // New Engl. J. Med. 2003. - Vol.349. - P.969-974.
Cao, Z. Association of matrix metalloproteinase-1 promoter polymorphism with generalized aggressive periodontitis in a Chinese population. / Z. Cao, C. Li, L. Jin et al. // J. Periodontal. Res. - 2005. - Vol. 40(6). - P.427-431.
Chung, CH Molecular portraits and the family tree of cancer. /
H. Chung, PS Bernard, CM Perou // Nat. Genet. - 2002. - Vol.32(Suppl). - P.533540.
Cirelli, JA AAV2/1-TNFR:Fc gene delivery prevents periodontal disease progression. / Cirelli JA, Park CH, MacKool K. et al. // Gene Ther. - 2009. - Vol.16(3). - P.426-36.
Colombo, AV Identification of intracellular oral species within human crevicular epithelial cells from subjects with chronic periodontitis by fluorescence in situ hybridization. / AV Colombo, CM da Silva, A. Haffajee et al. // J. Periodontal. Res. - 2007. - Vol. 42. - P. 236-243.
Colonel, V. Gene expression profiling of 12633 genes in Alzheimer hippocampal CA1: transcription and neurotrophic factor down-regulation and upregulation of apoptotic and pro-inflammatory signaling. / V. Colonel, J. Schurr, MJ Ball et al. // J. Neurosci. Res. - 2002. - Vol.70. - P.462-473.
Corbi, SC Clinical outcomes of periodontal therapy are not influenced by the ATC/TTC haplotype in the IL8 gene. / SC Corbi, LS Finoti, G. Anovazzi et al. // J. Periodontal Res. - 2014. - Vol. 49(4). - P.489-98.
Elamine, A. Prevalence of Aggregatibacter actinomycetemcomitans in Sudanese patients with aggressive periodontitis: a case-control study. / A. Elamine, JM Albandar, K. Poulsen et al. // J. Periodontal. Res. - 2011. - Vol.46(3). - P.285-91.
Hyvarinen, K. Detection and quantification of five major periodontal pathogens by single copy gene-based real-time PCR. / K. Hyvarinen, S. Laitinen,
S. Paju et al. // Innate immunity. - 2009. - Vol.15(4). - P.195-204.
Ianni, M. Variations in inflammatory genes are associated with periodontitis. / M. Ianni, G. Bruzzesi, D. Pugliese et al. // Immun. Aging. - 2013. - Vol.10(1). - P.39.
Inagaki, S. Porphyromonas gingivalis vesicles enhance attachment, and the leucine-rich repeat BspA protein is required for invasion of epithelial cells by “Tannerella forsythia” / S. Inagaki, S. Onishi, HK Kuramitsu, A. Sharma // Infect. Immun. - 2006. - Vol.74(9). - P.5023-5028.
Inagaki, S. Treponema denticola invasion into human gingival epithelial cells. / S. Inagaki, R. Kimizuka, E. Kokubu et al. // Microb. Pathog. 2016 Jan 22. pii: S0882-4010(16)00008-5. doi: 10.1016/j.micpath.2016.01.010.
Irshad, M. Cytokine and matrix metalloproteinase expression in fibroblasts from peri-implantitis lesions in response to viable Porphyromonas gingivalis. / M. Irshad, N. Scheres, D. Anssari Moin et al. // J. Periodontal Res. - 2013. - Vol.48(5). - P. 647-656.
Itagaki, M. Matrix metalloproteinase-1 and -3 gene promoter polymorphisms in Japanese patients with periodontitis. / M. Itagaki, T. Kubota, H. Tai et al. // J. Clin. Periodontol. - 2004. - Vol. 31. - P. 764-769.
Izuhara, K. Microarray-based identification of novel biomarkers in asthma. / K. Izuhara, H. Saito // Allergol Int. - 2006. - Vol.55. - P.361-367.
Javed, F. Cytokine profile in the gingival crevicular fluid of rheumatoid arthritis patients with chronic periodontitis. / F. Javed, HB Ahmed, T. Mikami et al. // J. Investig. Clin. Dent. - 2014a. - Vol. 5(1). - P.1-8.
Zhou, Q. Cytokine profiling of macrophages exposed to Porphyromonas gingivalis, its lipopolysaccharide, or its FimA protein. / Q. Zhou, T. Daste, M. Fenton et al. // Infect. Immun. - 2005. - Vol.73. - P.935-943.
Zorina, OA Analysis of the relationship of allele frequencies of the risk of aggressive periodontitis. / OA Zorina, OA Boriskina, NK Aymadinova, DV Rebrikov // The Way of Science. - 2014. - No. 6 (94). - R. 184-189.
Zorina, OA The relationship of genetic polymorphisms with the development of aggressive periodontites. / OA Zorina, OA Boriskina, NK Aymadinova, DV Rebrikov // Proceedings of the I International Scientific Conference "Global Science and Innovation". - Chicago, USA. - 2013. - R. 387-392
Downloads
Published
How to Cite
Issue
Section
License
This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.